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This was hypothesized by Jeremias and Gibson53 to induce too much wall pressure and immediate myofibrillar injury, resulting in cardiomyocyte Loss of life, and thus troponin release while in the absence of ischaemia (myocardial strain theory). This can be supported by both equally experimental scientific studies and scientific observations.is often

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Current American Higher education of Cardiology and American Coronary heart Association suggestions point out that troponin is the popular biomarker for diagnosing acute myocardial infarction (MI).1 Troponin T (TnT) And that i molecules have amino acid sequences which have been found only in cardiac tissue, creating their assays extremely specific

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